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Our data suggest activation of ferroptotic pathways could be one of the primary drivers of intracellular (and extracellular) redox imbalance in T2-high asthma

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In spinal cord injury, this peroxidation has been shown to precede mitochondrial dysfunction [83], while inhibition of lipid peroxidation with 21-aminosteroid in rats attenuated controlled cortical impact (CCI)-induced decreases in mitochondrial respiration [84]

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