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Liposomal encapsulation of butyrate effectively reduced its cytotoxicity while enhancing therapeutic efficacy [114]

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The mechanism is elegant

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A small fraction of an ingested dose is metabolized in the liver by the cytochrome P-450 mixed function oxidase enzyme system to form a reactive, potentially toxic, intermediate metabolite which preferentially conjugates with hepatic glutathione to form the nontoxic cysteine and mercapturic acid derivatives which are then excreted by the kidney

The long-term impact on weight loss goals depends on how quickly treatment is resumed and whether the interruption leads to sustained changes in eating behaviors