Source for direct demonstration of GLP-1R expression in HaCaT keratinocytes and that liraglutide upregulated keratinocyte migration via PI3K/Akt activation
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While GHSR1a activity in the hypothalamus is likely heavily involved in homeostatic mechanisms, its presence in extra hypothalamic regions, including a number of limbic and midbrain structures like the ventral tegmental area (VTA), suggest that this receptor is associated in processes associated with learning and motivation (Abizaid et al., 2006
We provide evidence that GSH indeed plays a key role in disulfide bond formation and redox homeostasis in the periplasm: We observed that GSSG can complement for the loss of DsbA and, unexpectedly, that GSH accelerates disulfide bond formation in the presence of DsbA
