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Its primary mechanism involves restoring the activity of key mitochondrial enzymes, such as SOD1, CCS, and CCO, thereby re-establishing metabolic function [51]

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Intern Med J 43: 100101, 2013 [DOI] [PubMed] [Google Scholar] 38.Kumar A, Bachhawat AK: A futile cycle, formed between two ATP-dependant -glutamyl cycle enzymes, -glutamyl cysteine synthetase and 5-oxoprolinase: The cause of cellular ATP depletion in nephrotic cystinosis

10.1097/MD.0000000000003629 Medicine