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Oxidative stress: the mitochondria-dependent and mitochondria-independent pathways of apoptosis

To determine whether dysregulated hepatic glutathione homeostasis and TNF can synergistically contribute to hepatocyte death, we performed cell culture studies using both human hepatoma cells and mouse non-transformed hepatocytes

Sensitivity analyses further confirmed the robustness of these findings, with stable associations across various OBS components (Supplementary Table S5)

ROS may disrupt cells or activate certain molecular and cellular pathways during cell death, promoting apoptosis

Every batch is independently tested by third-party laboratories