In addition, we treated PC cells with monensin to make SLC7A11 remain in Golgi and trace its expression upon monensin removal
Considering the therapeutic limitations of targeting individual phenotypes, future research should prioritize a more comprehensive approach to the diseases overall pathogenic processes
Liu P, Wu X, Gong B, L G, Li J, Gao H
2019 Jun 17;33:56
[DOI] [PubMed] [Google Scholar] 131.Oakley A.J., Lo Bello M., Battistoni A., Ricci G., Rossjohn J., Villar H.O., Parker M.W
A recent study found that under conditions of energy stress, such as glucose starvation and ATP depletion, activated AMPK is crucial for inhibiting erastin-induced ferroptotic cell death[38]