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The peptide works primarily by: Slowing gastric emptying to prolong satiety after meals Reducing food intake through central nervous system signaling Promoting satiety via actions at the area postrema in the brainstem Complementing insulin action without directly affecting insulin secretion For those exploring peptide research, understanding cagrilintide's unique mechanism is crucial for appreciating its potential applications in metabolic health studies

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However, these changes were not significantly different between them

They might be caused as a result of structural and chemical changes in the brain