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doi: 10.1371/journal.pone.0069362 160 ChengBZhangJShenQSunZLuoYHuY
While cumulative evidence has demonstrated that mitochondrial metabolism reprogramming plays crucial roles in the promotion of cancer progression, the molecular mechanisms underlying the alterations of mitochondrial metabolism in cancer cells remain incompletely understood
We previously produced the App NL-G-F/NL-G-F -KI ( App NL-G-F ) mouse strain using genomic DNA of introns 15 to 17 of mouse APP, which humanized the A sequence, and introduced KM670/671NL (Swedish), I716F (Iberian), and E693G (Arctic) mutations 41
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