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In vitro investigations have demonstrated that selenomethyl selenocysteine is capable of diminishing ROS levels, stabilizing the levels of prolyl hydroxylase-2 (PHD2) and prolyl hydroxylase-3 (PHD3), and downregulating HIF-1 expression in colon and head and neck cancer cells

Differences between the control and glutathione groups in the animal study were determined using Students t -tests

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Ongoing studies continue to examine cellular communication, angiogenic signaling, cell migration, cytoskeletal organization, extracellular matrix remodeling, and tissue homeostasis