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It combats dry eye syndrome and presents prominent anti-tumor potential. But there are significant caveats

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[7] [13] It has a half-life of approximately five days that supports weekly dosing

Therapeutically, p53 activation dualistically regulates apoptosis and ferroptosis to suppress PCa growth, but ferroptosis-driven macrophage polarization in the TME may inadvertently promote tumor metastasis [Figure 2A], emphasizing the need for context-specific modulation to balance efficacy and paradoxical outcomes

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