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The mechanism(s) involved in this unique pathophysiological feature of PKD is not understood

References: Adrian S, Scherzinger A, Sanyal A, Lake JE, Falutz J, Dub MP, Stanley T, Grinspoon S, Mamputu JC, Marsolais C, Brown TT, Erlandson KM

Most bronchi/bronchioles showed goblet cell hyperplasia/metaplasia associated with luminal inflammatory exudates (Fig

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An analysis of the mechanism reveals that the nanoparticles facilitate the oxidation of GSH into GSSG, leading to disruptions in redox balance and the antioxidant defense system and therefore accumulation of ROS and lipid peroxides