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FAD is then fully reduced to FADH 2 upon nucleophilic attack by Cys379 on the disulfide linkage between Cys201 and the FAD adduct, thereby reforming the initial trisulfide bridge

Therefore, targeting PRMT1 holds promise as a novel therapeutic strategy, particularly in kidney stone disease, in which metabolic derangement and inflammation are prevalent

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Their function is crucial for maintaining the balance of cysteine within cells, and disturbances in their function can result in health disorders