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Comprised of TXN, thioredoxin reductase (TrxR), and nicotinamide adenine dinucleotide phosphate (NADPH), this system facilitates the reduction of oxidative GSH to its active form in response to NADPH [78], thereby supporting the removal of cytotoxic GSH by GPX4 [79, 80]

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Oral administration of peptides faces hurdles related to gastrointestinal stability, however the invasive dosing routes lead to higher costs and lower compliance [2]

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The peak in rod degeneration in the rd1 mouse occurs between postnatal day (PN) 11 and PN13, and by PN20 all rods have degenerated 37