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Cultured fibroblasts also allowed studying the molecular interactions between potential chaperones and the -Gal A protein, thereby opening more alternatives for improvement of the treatments for Fabry disease [26]

Intriguingly, PIK3C3/VPS34 lactylation augments its interaction with Beclin1, Atg14L, and UVRAG, increasing its lipid kinase activity and promoting macroautophagy/autophagy and endolysosomal degradation

Adult humans have a relatively fixed number of fat cells (adipocyte turnover is slow, roughly 10% per year)

Ferroptosis antioxidant defense system mainly divided into GPX4-dependent systems (SLC7A11/GSH/GPX4 axis) and GPX4-non-dependent systems (FSP1-CoQ10-NAD(P)H axis, GCH1/BH4 axis, and DHODH/CoQH2 axis) [31, 32] (Fig

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Inflammatory mediators are suppressed as a result of their anti-inflammatory properties