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Our research findings suggested that SLC7A11, HMOX1, G6PD, RRM2, KIF20A, HELLS, GPT2, GLS2, SPP1, CCR2, DCN, IRS1, and IGF1 may play significant roles in HBV-ALF, and there were notable changes in the immune microenvironment in HBV-ALF

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Mechanistically, lactate increases the activity of adenosine monophosphate-activated protein kinase (AMPK) and suppresses the phosphorylation of serine/threonine kinase 1 (Akt) and mammalian target of rapamycin (mTOR) to prompt the apoptosis of DA neurons in PD [75] (Fig