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Building upon the natural hormone amylin, Cagrilintide is engineered with structural modifications to prolong its half-life and enhance receptor specificity [1]

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Across biochemical assays, cell culture models, and preclinical animal studies, mechanistic investigations have focused on how BPC-157 interacts with nitric oxide signaling, angiogenic regulation, cytoskeletal remodeling, and epithelial or connective tissue repair processes