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Mechanistically, oxidative stress triggers endoplasmic reticulum stress through the activation of the PERK-eIF2-ATF4 and IRE1-XBP1 signaling pathways, as well as through the upregulation of CHOP (C/EBP homologous protein), which promotes apoptotic signaling (75) as shown in Figure 4
Cheng, D., Yang, S., Zhao, X
Technology for improving the bioavailability of small molecules extracted from traditional Chinese medicines
doi: 10.3389/fphys.2020.00694 19 HeLHeTJiLLiuTMaX
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