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doi: 10.1152/ajpendo.00008.2008

Studies in rodent tendon fibroblasts have shown increased expression of VEGF (vascular endothelial growth factor) and EGF receptors following BPC-157 exposure, providing a biological basis for observed angiogenesis in wound models

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Potential direct mechanisms involve GLP-1 receptor activation on vascular endothelial cells and possibly immune cells, though human evidence for the latter is variable