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A thiol-disulfide transhydrogenase from yeast

Targeted therapy in nuclear medicinecurrent status and future prospects

To determine whether increases in intracellular GSH directly enhance expression of T2-signature proteins in HAECs, we lowered intracellular GSH levels by inhibiting SLC7A11 using the chemical inhibitor erastin (16)

In principle, a hyperoxidizing ER might simply blunt the UPR rather than improve ER function, since activation of at least the UPR sensors ATF6 and IRE1 can be inhibited by oxidation (Nadanaka, Okada, Yoshida, & Mori, 2007

T-dependent B cell immunization (LCMV Cl13 and VSV) Mice were intravenously infected with 2 10 6 pfu of LCMV Cl13

Wang TD, Tomas ND, Perera, et al