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Therefore, increased glutathione may enhance the blood oxygen carrying capacity and improve depleted glutathione stores, which hypothetically translates into enhanced performance outcome

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This counterintuitive finding suggested that while FOXO4 is involved in the senescence program, it also helps maintain the viability of senescent cells, potentially contributing to their accumulation [2]

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AKT1 participates in ferroptosis vulnerability by driving autophagic degradation of FTH1 in cisplatin-resistant ovarian cancer