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By activating telomerase and enhancing immune surveillance, the peptide can slow tumor growth and promote cellular apoptosis in malignant cells Anisimov et al., Neuroendocrinology Letters , 2003

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The research that does exist is consistently positive, and the safety profile looks excellent

[27] The connection between GSTP inhibition of the pro-apoptotic JNK pathway and the isozyme's overexpression in drug-resistant tumor cells may itself account for the tumor cells' ability to escape apoptosis mediated by drugs that are not substrates of GSTP