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Sun Y, Miao D, Zeng Z et al

In preclinical research models, glucagon receptor agonism has been shown to reduce hepatic fat content and increase resting energy expenditure, effects that are preserved when glucagon agonism is combined with GLP-1R and GIPR activation [3]

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Compared to ingredients that bank on trends, GHK-Cu has decades of research that support its role in skin repair, regeneration, and collagen production

As in the previously mentioned study there was no difference between GLP-1 receptor agonists and SGLT2 inhibitors in reducing the risk of MACE, but when compared to DPP-4 inhibitors SGLT2 inhibitors significantly reduced cardiovascular and total death

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