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Structure-toxicity relationship studies of amineptine gives a possible explanation of reactive metabolite formation by P450 enzymes upon NADPH-dependent microsomal covalent binding, suggestive of a characteristic reactive epoxide hepatotoxicant of amineptine (Geneve et al., 1987)

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1 These agents also delay gastric emptying and reduce appetite via central mechanisms, thereby contributing to their metabolic effects

The survival study was conducted as an exploratory pilot experiment to generate preliminary evidence of the therapeutic impact of IL1RAPCD3 in an AML xenograft model