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A small randomized, double-blind, placebo-controlled trial of IV glutathione in people with peripheral artery disease showed increased pain-free walking distance and improvement of macrocirculatory and microcirculatory parameters (Arosio E, et al

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Co-immunoprecipitation mass spectrometry and CRISPR knockout (KO) in mammalian cells identified mitochondrial m-AAA protease AFG3L2 as responsible for degrading SLC25A39 through the matrix loop 1

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