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Rats were fed with a standard rodent chow ad libitum

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Ongoing Monitoring and Support: Regular follow-up appointments track progress, allow treatment plan adjustments, and address concerns

The mechanism may be related to curculigoside significantly reducing AD-promoting factors (e.g., A1-42, p-tau) and increasing ferroptosis protective factors (e.g., GPX4, SLC7A11, GSH) in the hippocampus and cortex of AD mice, 574 suggesting that the natural compound curculigoside can be used as a promising therapeutic agent to improve AD by inhibiting ferroptosis

Apr 2019;7(4):1426-1437