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Lixisenatide inhibits neuroinflammation by promoting the proteasomal degradation of thioredoxin-interacting protein (TXNIP) in ocular tissues via the PI3K/Akt signaling pathway, an effect more potent than that of insulin

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In multiple clinical trials, SAMe has demonstrated an ability to reduce joint pain and stiffness with an efficacy comparable to non-steroidal anti-inflammatory drugs (NSAIDs), but without the severe gastrointestinal side effects often associated with long-term NSAID use

There is also an acute need of confirmation studies, especially regarding the clinical effects of cerebellar stimulation on human disease