Calcitonin can act on both amylin and calcitonin receptors
Gradual dissolution of NCs within tumor tissue releases Fe 2+ /Fe 3+ and Cu 2+ , which react with AS to generate carbon-centered radicals (C), amplify oxidative stress, deplete GSH, and down-regulate GPX4 and FDX1, thereby triggering ferroptosis and cuproptosis
Structural basis for flip-flop action of thiamine pyrophosphate-dependent enzyme relevated by human pyruvate dehydrogenase
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