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doi: 10.1016/j.celrep.2020.107776 118 GuttenplanK

2004, 82 (Suppl 3): 1171-1176

It was revealed through studies in uterine cancer models that caudatin stimulates TNFAIP1, which in turn inhibited NF-B transcriptional activity ( In vivo xenograft tumors further supported this mechanism of action, indicating that caudatin treatment of tumors led to increased levels of TNFAIP1 (Table 1, Row 11)

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10.1371/journal.pone.0035241 PLoS One 49 LeeC

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