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doi:10.1016/j.ajog.2016.07.040

Notably, overexpression of ubiquitin-specific protease 48 (USP48) can inhibit the above pathological processes by deubiquitinating SLC1A5, suggesting that targeting the ferroptosis-inflammation axis represents a promising potential strategy for DR treatment (168)

El dficit de acetilcolina colinrgica es el correlato bioqumico ms consistente del deterioro cognitivo asociado a la edad y est directamente implicado en la enfermedad de Alzheimer

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