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This temporal progression indicates that 5-amino-1MQ may serve as more than a simple enzyme inhibitor, potentially acting as a metabolic reprogramming agent that fundamentally alters cellular energy management strategies over extended periods

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Combinative treatment of beta-elemene and cetuximab is sensitive to KRAS mutant colorectal cancer cells by inducing ferroptosis and inhibiting epithelial-mesenchymal transformation

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Insulin/mTOR signaling was crucial for the differentiation of hematopoietic progenitor cells to granulocyte and monocyte/macrophage populations