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Release of pro-inflammatory mediators by microglia, the main effector cells of inflammation within the central nervous system (CNS), could be sufficient to cause AD pathology independently of other pathological events, such as deposition of the amyloid b protein (Ab) and neurofibrillary tangles (NFT) [4,5]

But glutathione amino acid precursors mitigated this effect, making it more useful than standard antioxidants to protect skin against environmental pollutants