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243 In AML, the level of H3K36me3 changes most dramatically before and after Met deprivation, and inhibition of STED2, the H3K36-specific methyltransferase, can reproduce most of the cytotoxic phenotypes produced by Met deprivation

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Even if this is a relatively small risk (which we do not know), how many of us practicing physicians would choose a therapeutic strategy for ourselves with insight into the potential for this risk

Corticotropin-releasing hormone deficiency reveals major fetal but not adult glucocorticoid need

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