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Nisin, an apoptotic bacteriocin, induces ChaC1 activation, calcium influx and cell cycle arrest in the G2 phase, leading to increased apoptosis and decreased cell proliferation in vitro and in vivo (68)

137 Blood Proteins as Antioxidants Human blood proteins are vulnerable to oxidative damage because free radicals can cause alterations in the electrical charge of proteins, induce cross-linking of proteins, and increase their susceptibility to proteolysis

4 Limitations and future directions Current evidence for H

Accumulated [ 3 H]acetyl-CoA was quantified by liquid scintillation counting

ROS elimination To maintain homeostasis, living organisms aim to control highly reactive ROS via antioxidant networks [15]

However, carefully selected tests may provide information about inflammation, atherogenic cholesterol particles, metabolic risk, lipid oxidation, and antioxidant status