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These effects typically occur during dose initiation or escalation and often improve as the body adapts to the medication

these synthetic GLP-1 receptor agonists (GLP-1RAs) mimic the effects of the natural hormone while overcoming its therapeutic limitations (such as its short half-life)

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In insulin-sensitive tissues, oxidative stress interferes with insulin receptor signaling through modification of critical cysteine residues and activation of stress kinases

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