An accumulation of excess intracellular oxidants can promote redox-mediated signaling events, including antioxidant responses, such as the activation of nuclear factor erythroid 2-related factor 2 (Nrf2) [3], as well as promote the activation of proinflammatory pathways that are mediated by redox-sensitive transcription factors, such as nuclear factor kappa B (NFB) and activator protein 1 (AP-1) [46]
Safety and efficacy of accelerated repetitive transcranial magnetic stimulation protocol in elderly depressed unipolar and bipolar patients
SSG of mitofusin 2 (MFN2) disrupts mitochondriaendoplasmic reticulum contact sites, compromising organelle communication and promoting activation of the RIPK1-RIPK3-MLKL axis in neurotoxicity models 82
Individuals with reduced-function variants may experience greater clinical benefit from supplementation than those with wild-type alleles
Derivatizations of agarose and polyacrylamide beads
Xiao Yao San improves depressive-like behavior in rats through modulation of -arrestin 2-mediated pathways in hippocampus
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