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The TGF-/Smad3 pathway is primarily regulated by progesterone during the fibrosis process of uterine fibroids [43,71]

Rapamycin exerts anti-ferroptotic effects by decreasing transferrin receptor expression, reducing cellular iron content, and activating the iron-regulated protein-1/2 (Irp1/2) system, potentially compensating for transferrin downregulation to maintain iron homeostasis (69)

MSC-Exos, which are nano-sized extracellular vesicles (30150 nm) loaded with bioactive materials such as proteins, lipids, and nucleic acids, have demonstrated significant therapeutic potential in tissue repair and immunomodulation [7,8,9]

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