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A Nuclear Magnetic-Resonance Study of Alamethicin-Induced, Delta-Hemolysin-Induced, and Melittin-Induced Sodium Leakage From Large Unilamellar Vesicles
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Underlying mechanisms of hepatic microcirculatory modulation included improvement in HSCs and LSECs phenotype (ATXi: desmin -24%, Vcam1 -28%
Esophageal Cancer Clinical Practice Guidelines
Assessment of mitochondrial mass did not reveal significant differences between NAFLD and control iPSC-Heps
It is not known whether antiviral therapy is also efficacious in groups with lower VL
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