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Malfertheiner et al

Research conducted in vivo and in vitro has shown that acrolein causes oxidative stress, resulting in the rupture of cell membranes, DNA and mitochondria damage, endoplasmic reticulum (ER) stress, and the potential to initiate apoptosis (112, 113)

There was no significant difference among the three groups in the vascular permeabilities on the non-ligature (contralateral) sides

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Moreover, current animal models are limited in their ability to fully replicate the complex pathological state of hypertension and lung cancer comorbidity, which hampers experimental design and interpretation