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In our study, the reduction of HDAC6 levels induced by oxidative stress led to migration and adhesion impairment, which was reversed by GLP-1 treatment

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FMT experiments further demonstrated that gut microbial dysbiosis enhances the intestinal permeability in microbiota-depleted mice and mediates intrarenal activation of NOD-like receptor signaling [135]

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In vitro glutathionylation with GSSG leads to glutathionylation of Cys119 and to the formation of the Cys167Cys190 intramolecular disulfide (Chan et al., 2016)