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Acknowledgements The authors acknowledge the logistic support and laboratory facilities of the Department of Biochemistry and Molecular Biology, Shahjalal University of Science and Technology, Sylhet, Bangladesh

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have revealed that in MDSCs derived from hepatocellular carcinoma patients, hypoxia activates HIF-1, leading to the upregulation of ectonucleoside triphosphate diphosphohydrolase 2 (ENTPD2) expression, which subsequently facilitates the enzymatic hydrolysis of extracellular ATP into 5-AMP