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Most predictive of poor clinical outcomes at 3 and 12 months were increases in LPCs (14:0, 16:0, 16:1, 17:0, 18:0, 18:2, 20:3 and 28:1), ketogenic AA, acylcarnitines, as well as BCAAs, NAA (n-acetylaspartate upregulates lipid degradation and fatty acid mobilization [48]), and tyrosine over the first four days after injury

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