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One possible mechanism is that ROS accumulation due to aberrant oxidative stress in erythroid cells affects erythropoiesis by inducing oxidative denaturation of the band 3 protein

Hemolysis has been clearly associated with emitting proinflammatory signals through releasing DAMPs [224]

BNC1 deficiency-triggered ferroptosis through the NF2-YAP pathway induces primary ovarian insufficiency

However, there is unmet need in the so called high risk population (huge tumor, bile duct invasion, main portal vein invasion) because efficacy of Ate+Beva is worse in those group

Victorino et al., 2014)

These two powerhouses create mutually beneficial effects that amplify their individual benefits