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Aliment Pharmacol Ther 57(3):313322

Substrates for these enzymes are drugs and xenobiotics, including warfarin , aflatoxin, adriamycin, chloramphenicol, and dimethylsulfoxide

Sci Rep, 2020

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Excessive activation of MR leads to increased expression of pro-inflammatory and profibrotic genes, and generation of reactive oxygen species, contributory to tubular inflammation and fibrosis ( Overall, the activation of RAAS in DKD results in renal tubular injury through inflammation, hypoxia, fibrosis, and altered sodium and water handling

Moreover, compared to wild-type mice, IRI or nephrotoxic insults led to more severe kidney injury and renal dysfunction in NRF2-deficient mice, as well as higher mortality [207]