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The authors reported that AOD-9604 stimulated lipolysis in isolated adipocytes and that chronic daily treatment in obese Zucker rats produced reductions in body weight gain of more than 50% relative to untreated controls over the treatment period, without effects on insulin sensitivity or IGF-1 levels [3]
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One method entails restricting replicative capacity particle assembly through deletion, such as UL55, UL56, US3, and UL23, thus confining resultant virulence strictly localized around affected sites (99, 102, 109)
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