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however, most patients are not candidates for these therapies and those that are, inevitably develop resistance to treatment (6)

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In human umbilical vein endothelial cells (HUVECs) and aortas from ApoE -/- mice, TMAO increased the concentrations of reactive oxygen species (ROS), especially superoxide, by inhibiting the SIRT3-SOD2-mitochondrial ROS signaling pathway (75)

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It explored the mechanisms of action of GLP-1RAs and their therapeutic potential in a wide array of diseases, such as diabetes mellitus, providing new insights into metabolic disease management

Interleukin-1 induces an inflammatory response and the breakdown of the endothelial cell layer in an improved human THBMEC-based in vitro bloodbrain barrier model