At the excitation of such red-edge molecules, their surrounding solvent cannot fully relax before fluorescence emission occurs, leading to the red-shift of the emission spectrum compared to the emission observed when exciting at the absorption maximum (Demchenko, 2002)
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This leads to the stiffening of arteries, increased blood pressure, and inflammation, all of which contribute to cardiovascular complications
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(P)RR knockout, even tissue-specific, is lethal compared to other RAS components knockout, suggesting that (P)RR has an important function independent of (pro)renin
9tv) revealed that in the M group, the percentage of M1 macrophages was significantly increased (36.78% in M and 28.41% in CTR), and the percentage of M2 macrophages was significantly reduced (24.15% in M and 29.95% in CTR), whereas anti-CXCL10 antibody treatment significantly reduced the percentage of M1 macrophages (29.85% in CM) and increased the percentage of M2 macrophages (29.89% in CM) close to the control in the CM group
