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The rationale was that direct delivery to the joint would maximize local efficacy while minimizing systemic exposure

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However, we tried several values of V mean and V eff as well as replacing the ohmic formalism by the GHK formalism and every version of the model gave a qualitative synergy

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In a mouse model of pressure-overload heart failure, the inhibitor significantly alleviated myocardial remodeling and dysfunction by inhibiting H 2 S degradation, demonstrating the feasibility of targeting the H 2 S metabolic pathway for treating heart failure

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