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For tumours that are significantly LDH-dependent, as described above, genetic or pharmacological inhibition of LDHA effectively reduces cancer cell glycolytic flux and NADH/NAD ratio, and results in cancer cell apoptosis

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Protective effects of catalpol and rhein in murine experimental autoimmune encephalomyelitis via regulation of T helper (Th) 1, Th2, Th17, and regulatory T cell responses

Blots were developed using Immobilon Forte Western HRP substrate (Sigma, WBLUF0100) and imaged with Microchemi 4.2 (BioRad)

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