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To establish a causal link between CeA Glp1r neuron activity and mesolimbic dopamine output, we selectively expressed human GLP1R in the CeA of Glp1r-Cre mice and monitored NAc dopamine dynamics during HFD consumption after administration of danuglipron or orforglipron (Fig

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The "kill" mechanism of Adipotide suggests a more permanent alteration to the animal's physiology, as the body does not rapidly regenerate destroyed adipose vasculature

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Taurine has been shown to facilitate nerve blood flow, motor nerve conduction, and nerve sensory thresholds in diabetic neuropathic rats

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