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Oxidative stress markers in bipolar disorder: a meta-analysis

As predicted, antimycin A significantly inhibited complex II-III activity

Cd8+ T cells and fatty acids orchestrate tumor ferroptosis and immunity via acsl4

Kikuchi M, et al

This phenomenon, similar to what has been proposed in Parkinsons disease [46], suggests a compensatory mechanism where cells increase transcription to compensate for reduced GPX4 protein levels

They catalyze the conjugation of a diverse range of electrophilic compounds to reduced glutathione (GSH), thereby playing an important role in the metabolism of xenobiotics including drugs, herbicides and pesticides [1, 2]